3. MP evidence of remission and/or cure...

(note: You may find it easier to follow this series of articles by using the Archive index on the right hand side of this page. I'm numbering the entries so you can 'click' each one in sequence.)

Here is some preliminary data from Dr. Marshall regarding the response-rate of patients with various manifestations of Th1-Inflammatory Diseases. While Sarcoidosis was the original thrust of the research and protocol, the evidence began to suggest that others might also benefit from the protocol. This has largely proven to be true as folks with various chronic illnesses have reported progress - often very dramatic. Others have struggled, especially in the beginning, seeing no sudden improvements or even a worsening of symptoms. This is explained by Dr. Marshall as the difficult adjustments our bio-chemistry and hormonal systems must go through as the bacteria are killed.

My personal experience seems to bear this out. More about that later. For now, here are two more slides:
1. Patients on protocol / vs. patients experiencing recovery (remission? cure?)



2. This slide is very interesting as it reveals some surprising resolutions of other conditions that were not originally expected. It does lend some credence to the idea that intra-cellular bacterial infection is more wide-spread and causing more illness than we realized:

4. Do the 'bugs' make us ill?

(note: You may find it easier to follow this series of articles by using the Archive index on the right hand side of this page. I'm numbering the entries so you can 'click' each one in sequence.)

Here's a quote and a link to the larger discussion about how 'occult' bacteria may be causing illness. There is a growing body of evidence explaining how sub-microscopic bacterial forms interfere with normal cell biology. They apparently have evolved a means of insinuating themselves into the cell's chemistry for their own survival, and thereby interfere with the normal inflammatory processes - (normally inflammation is part of our healing process).

I'll post more on this later...

http://www.marshallprotocol.com/forum37/3391.html

These remissions also resolved a second issue, whether such intra-cytoplasmal bacteria were pathogenic or benign. In the case of Sarcoidosis, the coccoid bacterial forms proved pathogenic. Two recent in-vitro studies have described how a similar species of intracellular bacteria directly modulated the transfer factor Nuclear factor-kappaB (NF-kB) in the cytoplasm, initiating a release of mRNA (signaling cytokine release) from the Nucleus. This is likely to be the pathogenic mechanism, and we have described how this mechanism can fuel the granulomatous inflammation of sarcoidosis, an inflammation which is not driven by normal lymphocyte-phagocyte signaling, but by a steady release of cytokines independent of any lymphocytic intervention.